Vaginal Discharge Solution
Reported 12 vaginal or urethral erosions in 1715 patients who underwent autologous pubovaginal sling placement (0.007 ) compared with 41 erosions in 1515 patients treated with various synthetic slings (0.027 ).1 The mechanism of erosion is thought to be secondary to delayed infection of synthetic materials resulting in vaginal erosion, and excessive tension applied to the sling or unrecognized urethral injury at the time of operation resulting in urethral erosion. The symptoms of erosion may include vaginal and urethral pain, irritative voiding symptoms, vaginal discharge, or bleeding. Erosions can be detected by physical examination and urethroscopy.
The pH level can be determined by placing pH paper on the lateral vaginal wall or immersing the pH paper in the vaginal discharge. A pH level greater than 4.5 often indicates the presence of bacterial vaginosis. It may also indicate the presence of Trichomonas vaginalis.
A 25-year-old woman is 2 weeks beyond her estimated date of delivery. She reports no pain, no labor contractions, no vaginal bleeding, no leaking fluid from her vagina, and no vaginal discharge. She reports that her fetus is moving. On further history, you find that the patient reports no other complaints, and her medical, surgical, social, and family histories are all negative. The physical examination you perform produces normal findings. Notably, her uterine fundal size measurement is 40 cm. Her pelvic examination reveals that her cervix is 3 cm dilated, 50 effaced, soft in consistency, and midpo-sition in the vagina. The fetal station is presenting at 0. You find no evidence of ruptured membranes. Uterine monitoring shows no contractions. You and the patient decide that labor induction would be safe and appropriate. What would be a good course of action
Vaginal discharge, if present, is usually thick and adherent, with white curds. 3. Other causes. In sexually active adolescents, the most common causes include bacterial vaginosis ( fishy, foul-smelling discharge), candidal vulvovaginitis (white, cottage-cheese'-appearing discharge), and trichomoniasis (malodorous yellow, frothy discharge).
Common nonvaginal etiologies include contact dermatitis from spermicidal creams, latex in condoms, or douching. Any STD can produce vaginal discharge. B. There is usually little itching, no pain, and the symptoms tend to have an indolent course. A malodorous fishy vaginal discharge is characteristic. The odor, a result of anaerobic bacteria, is exacerbated during menses and following intercourse due to the alkaline nature of blood and semen. C. There is usually little or no inflammation of the vulva or vaginal epithelium. The vaginal discharge is thin, dark or dull grey, and homogeneous.
Prevention of Inflammatory eye disease in the newborn Information from the Maternity Clinic Leipzig by Crede [22
I am therefore convinced ( ) that all affected children in ( ) hospital ( ) were infected solely by direct transmission of vaginal discharge to the eye during delivery. The infected eye usually begins to show symptoms of disease 2 or 3 days after birth, but also sooner or later - the sooner, the more serious the condition. In the following part, Crede stated that it was his achievement, having obtained the insight that vaginal douches were almost ineffective and that the contagious agent had to be destroyed sufficiently, that the prophylactic efforts, which had not been performed before, were put into place. As a method for the sufficient destruction of the contagious agent he stated again the administration of 2 silver nitrate solution directly into the eyes of every newborn child, including consecutive hygienic precautions to prevent a later inoculation of the child's eye by vaginal discharge from the mother.
Trichomonas vaginalis is a common sexually transmitted pathogen causing vaginitis, exocervicitis, and ureteritis in women (Fauts and Kraus 1980). Trichomonas vaginalis infections has been suggested to play a role in the pathogenesis of pre-term birth, pre-term rupture of membranes and delivery of low-birth-weight infants (Cotch et al. 1991 Read and Klebanoff 1993). Recently T. vaginalis infections has been implicated as a cofactor in the transmission of HIV (Laga et al. 1993). Trichomonas vaginalis infection are frequently asymptomatic, and early, accurate diagnosis are required for specific treatment. Routine diagnosis of T. vaginalis usually depends on direct microscopic identification of the parasite in wet mount preparations. However, wet mount examinations detects only 60 and the direct immunofluorescense using monoclonal antibodies detect 86 of culture positive cases in women. Although culture is considered the most reliable diagnostic method, with a sensitivity of 90 for...
In postpubertal girls, when was the last period Is there a history of sexual intercourse Vaginal discharge Consider pelvic inflammatory disease (PID), mittelschmerz, and ectopic pregnancy, depending on gynecologic history. K. Fever With appendicitis, there is typically afebrile or low-grade fever until perforation with viral conditions and peritonitis, temperature may be highly elevated. L. Chronic systemic illnesses Children with sickle cell disease may have abdominal pain from a crisis those with diabetes can have abdominal pain associated with ketoacidosis. Leukemia may produce typhlitis during periods of severe leukopenia. Inflammatory bowel disease can cause abdominal pain during periods of exacerbation.
Vulvovaginal thrush is a relatively common problem, representing roughly a quarter of all infectious vaginitis, with a large proportion of women experiencing at least one episode of vaginal thrush during their lifetime (Sobel et al., 1998). Some women experience multiple episodes, commonly called recurrent vulvovaginal candidiasis (RVVC). Symptoms of vulvovaginal candidiasis include itching, burning, soreness, and abnormal vaginal discharge. In women presenting with vulvovaginal candidiasis 60-100 of isolates are identified as C. albicans (Giraldo et al., 2000 Lopes Consolaro et al., 2004 De Vos et al., 2005 Beltrame et al., 2006 Moreira & Paula, 2006 Paulitsch et al., 2006 Pirotta & Garland, 2006).
Duration, characteristics, and severity of the incontinence, precipitating factors and reversible causes should be assessed. Dysuria, urgency, pelvic pain, dyspareunia, constipation, fecal incontinence, pelvic prolapse, or abnormal vaginal discharge should be sought. A history of diabetes, thyroid disease, spinal cord injury, cerebral vascular accidents, urethral sphincter
The 1-hour pad test is usually performed in the office. The patient's bladder is prefilled, either with a catheter or by having the patient drink water, until it feels full or a set volume is reached (i.e., 250mL). The patient then performs a standard set of activities for the hour. These activities must be representative of routine daily life such as bending, coughing, and climbing stairs. The pad must be weighed before and after the test and the weight gain represents the volume of fluid loss. Every effort should be made to prevent drying of the pad. Ideally,waterproof underclothing should be worn during the test period and, once removed, the pad should be sealed in a plastic bag until it is weighed. It is important to note that a weight gain of up to 1 g over an hour can be accounted for by sweat and vaginal discharge.
Sensitive to estrogen deprivation, and symptoms may appear promptly as soon as estrogen levels begin to decline. Vaginal atrophy will present with a variety of symptoms along a continuum of severity. Symptoms are typically vaginal dryness and associated dyspareunia, which may be initially presented as vaginal irritation. This will progress to loss of vaginal rugation and development of a progressively pale, hypovascular mucosa. Eventually, a thin inflammatory exudate can develop, sometimes in large quantities. This may result in a watery vaginal discharge that on microscopic examination is replete with inflammatory and basal squamous epithelial cells, but with no evidence of bacterial infection. As with other more obvious mucosal hypoestrogenic states, a chronic watery vaginal discharge without an infectious cause in a postmenopausal woman warrants a course of local estrogen therapy. Up to this stage of atrophy progression, resultant changes are readily reversible with local estrogen...