How to get rid of striae naturally

Stretch Marks

Stretch Marks

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4.6 stars out of 11 votes

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Childhood Glaucomas Definition

Goniotomy And Trabeculotomy

The cornea will appear whitish and opacified due to epithelial edema. Breaks in Descemet's membrane can exacerbate an epithelial or stromal edema. These lesions, known as Haab's striae, will exhibit a typical horizontal or curvilinear configuration. Striae in Descemet's membrane. In contrast to the horizontal Haab's striae in congenital glaucoma, endothelial breaks can also occur as a result of injury during a forceps delivery (vertical striae), in keratoconus, and in deep ker-atitis.

The psychiatry of pregnancy Pregnancy adjustment

Her change in appearance and shape is sometimes distressing to the mother. Some take pride and pleasure in their pregnant appearance, enjoy the extra attention, and feel an enhanced sense of womanliness. Others are concerned about their loss of figure and facial bloom, weight gain, and potential stretch marks.

Ipsilateral corticocortical association connections

The olfactory pathway is unique among the sensory modalities in having direct access to the cerebral cortex without passing through the thalamus. The primary olfactory receptor neurones of the olfactory mucosa send their axons (the fila olfactaria) through the cribriform plate of the ethmoid bone directly into the overlying olfactory bulb, where they contact the mitral cells in synaptic glomeruli. Axons of the mitral cells pass caudally in the olfactory tract to the anterior perforated substance. Here the olfactory tract splits into medial and lateral olfactory striae. All the mitral cell axons pass in the lateral stria. The medial stria contains axons mainly from the anterior olfactory nucleus, which are destined for the contralateral olfactory bulb by way of the anterior commissure. The lateral olfactory stria passes to the medial temporal lobe, where the axons terminate in the anterior margin of the entorhinal cortex, the pyriform cortex, and the corticomedial subdivision of the...

Corneal Opacifications

Corneal Opague Girl

Much attention is focused on the cornea in the diagnosis of congenital glaucoma. There are several typical signs that can be observed such as a stretched superior limbal region (Fig. 7.3), tears in the Descemet membrane, the so-called Haab striae (Fig. 7.4), and corneal opacifications (Fig. 7.5). Corneal opacities are frequent in infantile glaucoma, occurring in up to 75 of glaucomatous eyes 17 . Corneal clouding as an isolated ocular symptom (without Haab striae and limbal stretching) in infants is a suspect sign of a genetic (congenital hereditary endothelial dystrophy, Turner or Noonan syndrome) Fig. 7.4. Haab striae (endothelial tears) in a adult patient who had undergone glaucoma surgery for congenital glaucoma in his first year of life Fig. 7.4. Haab striae (endothelial tears) in a adult patient who had undergone glaucoma surgery for congenital glaucoma in his first year of life

Behavioural phenotypes

Signs of connective tissue dysplasia such as hyperextensible finger joints, flat feet, inguinal and hiatus hernia, enlarged aortic root, and mitral valve prolapse can be detected in some cases. Foot abnormalities such as flat feet, a sandal gap, and long broad toes with recurrent paronychia of the first toes have also been reported. In a certain number of adult males, hypogonadal appearance with decreased body hair, gynaecomastia, and striae have been reported. Other occasional features associated with fragile X syndrome include torticollis, kyphoscoliosis, pectus excavatum, hyper-reflexia, and nystagmus. Epilepsy is reported in about 25 per cent of these individuals. In some cases an abnormal response of thyroid-stimulating hormone release in response to administration of thyrotropin-releasing hormone has been reported. Other medical conditions such as strabismus, otitis media, sinusitis, joint dislocation, orthopaedic problems, and apnoea may also affect subjects with fragile X...

Biology and nomenclature

Nomenclatura Parasitos

The anisakid larvae found in fish are all third stage. Lips are absent, but a small boring tooth is present on the head, and there are no visible reproductive organs. The shape of the head, tail, and anterior digestive tract is used for distinguishing each larva. Anisakis simplex (Figure 16.1) forms a 3-4 mm tight flat spiral, while P. decipiens (Figure 16.2), being larger and yellowish-brown, is shaped more like a corkscrew. Both have a glandular ventricle, but P. decipiens also has an intestinal caecum. Contracaecum Phocascaris larvae have opposed intestinal caecum and ventricular appendix, and near the head, the cuticle has characteristic transverse striae and longitudinal ribs, giving it a tessellated appearance (Figure 16.3). Similar drawings are found in the literature, but to actually see these characters in whole worms through a microscope, fixed dead specimens should be made transparent by clearing them in lactic acid, glycerol, phenol-ethanol, or lactophenol.

Olfactory System Structure And Organization

The olfactory system is composed of the olfactory epithelium, the olfactory nerves, the olfactory bulbs, the olfactory tracts, and the median and lateral olfactory striae that terminate in the contralateral hemisphere or the ipsilateral amygdaloid nucleus, septal nuclei, and hypothalamus. The olfactory epithelium is located on the superior-posterior aspect of the nasal septum and the lateral walls of the nasal cavity and contains the olfactory sensory neurons (OSNs). The OSNs are generated in situ from stem cells. Aging OSNs are replaced by cell division that persists into adulthood and throughout the adult life. The life span of an OSN is in the range of weeks to months. The OSNs are bipolar neurons, the axons of which form the olfactory nerves and pass through the cribiform plate and terminate in the olfactory bulb, where they synapse with second-order neurons and interneurons. In the olfactory bulb, the OSN axon terminates in a glomerulus, which is a globoid neural structure. The...

Nonmalignant Skin Disease

The most common and debilitating skin problems arise from the long-term use of glucocorticoids. The manifestations include striae, friable skin, ecchymosis, acne-form rash and cushingoid features. In addition, cyclosporine may cause thickening of the skin, hypertrichosis, gingival hyperplasia, epidermal cysts, pilar keratosis, and folliculitis. Treatment includes reducing the dosage of these medications to the lowest level necessary to maintain immunosuppression and topical measures.

Carbonic Anhydrase Inhibitors

Systemic acetazolamide may cause severe systemic side effects including metabolic acidosis, renal problems, and hepatic necrosis. Owing to its chemical structure, acetazolamide should not be given in cases with a sulfonamide allergy. Under controlled circumstances, acetazolamide is sometimes used for short periods, preferably in older children. Topical carbonic anhydrase inhibitors, such as dorzolamide and brinzo-lamide, are less likely to cause side effects than oral acetazolamide, but seem to be less effective in pressure reduction 45 . Little is known about the disturbance of endothelial function in buphthalmic eyes with extensive Haab striae on treatment with topical carbonic anhydrase inhibitors.


Tacrolimus is a macrolide lactone originally derived from Streptomyces tsukubaensis. Although structurally unrelated to cyclosporine, tacrolimus has a very similar mechanism of action that is, it blocks the production of proinflammatory cytokines by T lymphocytes by inhibiting calcineurin. Tacrolimus, however, appears to be 10 to 100 times as potent as an immunosuppressive. Oral tacrolimus (FK506) is used for prevention of organ rejection in recipients of renal and hepatic transplants. A topical formulation (Protopic) has recently been approved for treatment of moderate to severe atopic dermatitis in children and adults who have not responded to other therapies. Levels of systemic absorption are low even when applied to a relatively large body surface area. Local irritant reactions (burning, stinging, erythema) are a common side effect, but these usually resolve within the first few days of treatment. The major benefit of topical tacrolimus over topical corticosteroids is that...

Ocular Examination

Juxtapapillary Hemangioma

Chorioretinal folds appear as a series of delicate striae, which are most often present in the posterior pole (Figure 6.2). Lines are usually parallel, but rarely they may radiate haphazardly to all directions.5 Although chorioretinal folds are most commonly seen with orbital tumors, they are also seen with muco-celes and other types of cysts and also in cases of orbital injury.6-9 Most chorioretinal folds are without symptoms and do not affect visual acuity. FIGURE 6.2. Fundus photograph showing horizontally aligned choroidal striae in a patient with intraconal cavernous hemangioma. present without scleral indentation, and in other patients, rapidly expanding orbital tumors lead to scleral indentation without chorioretinal folds.11 There is no clear relationship between the size of the space-occupying lesion and the extent or direction of the chorioretinal folds. Furthermore, the position of the chorioretinal striae is not a very dependable finding on which to localize the...


Lar elevation associated with a pit of the optic nervehead in his right eye. He had received laser treatment four months earlier On examination, his visual acuity in this eye w7as 20 50. Slit-lamp biomicroscopy (A) revealed a pit along the inferotemporal rim of the optic disc, a small cyst directly in the fovea, fine superficial retinal striae, and an underlying vellow discoloration of the pigment epithelium.

Lichen Planus

Sions are frequently asymptomatic, but in some patients lichen planus can lead to intractable oral ulceration that may persist for decades. The lesions have characteristic clinical appearances and distribution. The most common form is striae, which are sharply demarcated and form lace-like (reticular) or annular patterns. These may be interspersed with defined, small, elevated papules. The patient may complain that they feel a slight restriction on opening. Less common types of white lesions are confluent plaques, which some term homogeneous lichen planus. They are usually well demarcated, raised plaques and are frequently traversed by intersecting grooves producing a tessellated appearance. The latter appearance is particularly common on the dorsum of the tongue and other sites in long-standing disease. Atrophic areas, with redness due to mucosal thinning but without ulceration are usually combined with areas of striation. Erosions are shallow, irregular ulcers usually covered by a...

Cushings syndrome

Hypersecretion of cortisol by the adrenal gland can result in Cushing's syndrome. Cushing's syndrome can also be due to exogenous ACTH or glucocorticoid administration, or endogenous hyperproduction of these hormones. Because a physiological release of cortisol occurs during periods of stress or duress, it is not surprising to see elevations of serum cortisol during the courses of many psychiatric disorders, including major depressive disorder, alcoholism, anorexia nervosa, panic disorder, and psychoactive substance-withdrawal syndromes. The more common clinical signs and symptoms of Cushing's syndrome include fat redistribution, menstrual irregularities, dysphoria, thin skin, moon facies, increased appetite, sleep disturbances, hypertension, hypercholesterolaemia, hypertriglyceridaemia, poor concentration, impaired memory, euphoria, glucose intolerance, striae, and hirsutism. (73