The aetiopathogenesis of Lewy bodies is poorly understood. They presumably form from an interplay between environmental stimuli and the intrinsic responsiveness of neurones.(5) Dementia with Lewy bodies, like Parkinson's disease, does not appear to have a strong heritable component, although the possession of an apolipoprotein e4 allele appears to confer a risk of developing DLB rather than Parkinson's disease. An inactive allelic variant of cytochrome oxidase (CYP2D6B) is more frequent in Parkinson's disease than controls, but there is disagreement whether the same is true for DLB.

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