Effective Home Remedy to Cure Flatulence and Bloating

Ultimate Flatulence Cure

Ultimate Flatulence Cure is a complete instructional program that helps you cure your bad stomach gas, bloating, excessive burps of flatulence problems. Joseph Arnold is the author of this program. He is also a medical researcher, a health consultant, and he suffered from this disease for 3 years. In the program, people will discover a list of foods they must avoid in the flatulence treatment process. Besides, the program reveals to users ways to lose weight, and ways to reduce some skin problems. The program is designed to be suitable for those who want to eliminate their flatulence without any medication. In combination with the guides which come packaged with it as a set of free gifts, this book will not only give you the tools to cure your own flatulence, it will also give you important tips which will allow you to feel better in a number of different ways. Instead of wasting money on medications which may work as a stop-gap, taking heed of this guide will allow you to see benefits above and beyond the stopping of symptoms.

Ultimate Flatulence Cure Summary


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Contents: 55 Page EBook
Author: Joseph Arnold
Price: $27.00

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Fibric acid derivatives

Raise HDL levels by up to 20 percent, and reduce LDL levels by approximately 5 to 15 percent.Gemfibrozil (Lopid) is particularly useful in patients with diabetes and familial dysbetalipoproteinemia. 2. Side effects of gemfibrozil include nausea, bloating, flatulence, abdominal distress and mild liver-function abnormalities. Myositis, gallstones and elevation of the LDL cholesterol level have also been reported. Fibrates should generally not be used with HMG-CoA reductase inhibitors because the risk of severe myopathy.

Clinical features

Acute intestinal ischemia most commonly presents with severe abdominal pain out of proportion to objective physical findings. This deceptive clinical picture may last for a few hours to several days. The pain is usually periumbilical but may sometimes occur in the right upper quadrant or right iliac fossa. With an embolus the pain usually occurs suddenly, while in arterial and venous thrombosis or non-occlusive ischemia the onset may be more insidious. Occasionally, particularly in non-occlusive infarction, pain may be absent altogether and the conspicuous clinical feature is silent abdominal distension. More often, this is preceded by intense peristaltic activity resulting in spontaneous evacuation of the bowel. Bloody diarrhea is not evident until several hours into the course of the disease when mucosal infarction has begun, but the stools may test positive for occult blood at an early stage. Physical findings are few. Bowel sounds may be hyperactive or normal. Tenderness is mild...

Recognition of complications

Bacterial or fungal infection of devitalized areas inside and outside the pancreas is responsible for 80 per cent of deaths from severe acute pancreatitis. Early identification is crucial, since drainage and often surgical debridement are mandatory. The associated mortality is directly related to any delay in diagnosis and proper treatment. Persistent remote organ failure or unexpected clinical deterioration (epigastric pain, vomiting, abdominal distension with guarding or rebound) should raise the suspicion of regional infection and prompt appropriate investigation. Clinical and biochemical indicators of pancreatic infection include the combination of fever exceeding 38.5 C, base excess above 4 mmol l, and hematocrit below 35 per cent. However, precise differentiation between inflammatory sterile and infected collections cannot be made reliably on clinical and laboratory grounds. Fever and leukocytosis are absent in 15 to 20 per cent with infected pancreatic necrosis or pancreatic...

Prognosis and predictive factors

High-grade, and nonmucinous histology 345, 1365, 1769 . The spread of mucus beyond the right lower quadrant of the abdomen (whether or not cells are identified within it) is an independent prognostic variable, as is the presence of neoplastic cells outside the visceral peritoneum of the appendix 250 . When pseudomyxoma peritonei is present, abdominal distension, weight loss, high histological grade, and morphological

History and Physical Examination

The patient is a 40-year-old G2P1 woman with a 4-year history of stress urinary incontinence (SUI) and fecal incontinence (FI) that began with the vaginal delivery of her now 4-year-old daughter complicated by a fourth degree laceration. Since that time, she has had progressively worsening FI to both liquid and solid stools, as well as an inability to control flatus. Her urinary incontinence is stress related with no significant urgency, frequency, enuresis, or nocturia. She voids with a normal flow. In addition, she complains of dyspareunia and vaginal dryness. Her symptoms have caused significant quality-of-life impairment.

Effects on pulmonary function

Abdominal trauma influences pulmonary function. Diaphragmatic tears and abdominal wall injuries have a direct effect on muscles used for inhalation. Rapid superficial breathing due to abdominal pain leads to hypoventilation, with atelectasis and pneumonia. Expectoration is decreased after a laparotomy. Abdominal distension leads to increased pressure on the diaphragm, with subsequent decrease of vital capacity followed by atelectasis.

Spectrum of clinical presentations and associated conditions

The classical presentation of celiac disease includes diarrhea, failure to thrive or weight loss, abdominal distention, flatulence and fatigue. However, during the past two decades there has been an increasing awareness that there are many less dramatic or classical presentations of celiac disease including iron-deficiency anemia, folate deficiency, osteopenic bone disease, as well as relatively non-specific gastrointestinal (GI) symptoms (Table 6.2). These so-called atypical forms of celiac disease are now more common than classical celiac

Anesthetic management

Anesthetic management in patients with acute cervical spinal cord injury may require the following monitoring and instrumentation electrocardiography arterial catheter to measure arterial blood pressure pulmonary artery catheter to measure cardiac output, pulmonary artery pressure, and pulmonary artery wedge pressure sophisticated mechanical ventilation devices end-tidal CO 2 pulse oximetry blood gas analysis somatosensory evoked potentials temperature devices nasogastric tube to reduce abdominal distension and risk of aspiration, and to measure gastric pH urinary catheter warming devices. The optimal technique of endotracheal intubation in patients with cervical spinal injury is related to the condition of the patient and his or her level of co-operation, the neurological deficit, and the training and experience of the individual performing the intubation. In conscious normoxic normocapnic patients without head injury, awake elective nasotracheal intubation may be performed following...

Immediate Questions

Is patient passing flatus or stool What is the character of the stools Often, decreased NG output correlates with return of bowel function. Abdominal obstruction or ileus may result in decreased passage of gas or bowel movements. Fresh red blood from the rectum along with bloody NG drainage is very serious. Melena suggests upper tract or small bowel bleeding. Stools that are normal in appearance and occult blood-positive are suggestive of slower GI bleeding. Stools that are negative for occult blood suggest very early or insignificant bleeding.

Emeticsinducing Vomiting

In cases where vomiting is contraindicated, the patient should be administered activated charcoal, which is available in tablets, capsules, or suspension. Charcoal absorbs (detoxifies) ingested toxic substances, irritants, and intestinal gas. Activated charcoal can be given as a slurry (30 grams in at least 8 oz. of water) or 12.5-50 grams in aqueous or sorbitol suspension. It is usually given as a single dose.

Components and Nutraceutical Characteristics of Soybean

Acids, i.e., healthful oil Mature soybeans are mainly raffinose (0.1-0.9 ) and stachyose (1.4-4.1 ), whose presence is associated with the flatulence produced after human consumption moreover, oligosaccharides are powerfull prebiotics and have been successfully commercialized in Japan for years 12 isomers of daidzein, genistein, and glycitein (aglycones), transformed to daidzin, genistin and glycitin after glucosilation isoflavon content is 1-4 mg g d.w. isoflavones inhibit the growth of cancer cells, lower cholesterol levels, and inhibit bone resorption

Fecal Incontinence Qualityof Life Scale

Fecal incontinence can dramatically disrupt the lives of individuals who have this debilitating condition. The inability to control the passage of stool or flatus can produce embarrassment, and the fear of such episodes may severely limit daily activities. Therefore, one measure of the Incontinence for flatus scores 1-3, incontinence for liquid scores 4-6, incontinence for solids scores 7-8 Incontinence for flatus scores 1-3, incontinence for liquid scores 4-6, incontinence for solids scores 7-8, lifestyle alteration scores 10-12

Concept and diagnostic entity

Eventually it became clear that the expanding kaleidoscope of symptoms should be managed in a way that made some sense. Beard approached this problem by organizing the symptoms into subtypes of neurasthenia cerebrasthenia (cerebral exhaustion) characterized by symptoms that were directly or indirectly connected with the head myelasthenia (spinal exhaustion) was defined by symptoms related to the involvement of the spinal cord digestive asthenia was characterized by dyspepsia, constipation, and flatulence. As time went on more subtypes were added by other investigators and specific treatment approaches were developed. The first list of symptoms Freud proposed for neurasthenia proper included headache, spinal irritation, dyspepsia with flatulence, and constipation. Later, he added sexual weakness and fatigue.

Pharmacologic options

Orlistat is a synthetic hydrogenated derivative of a naturally occurring lipase inhibitor, lipostatin, produced by the mold Strepto-myces toxytricini. Taken at a therapeutic dose of 120 mg tid, orlistat blocks the digestion and absorption of about 30 of dietary fat. Recently, the XENDOS trial randomized 3,305 obese subjects to lifestyle changes, plus either 120 mg of orlistat or placebo 41 . After 4 years, orlistat plus lifestyle changes led to a 37.3 (P 0.0032) risk reduction in the development of diabetes and improved weight loss compared with placebo plus lifestyle changes. Multiple randomized, 1- to 2-year double-blind, placebo-controlled studies have shown that after 1 year, orlistat produces a weight loss of about 9-10 compared with a 4-6 weight loss in the placebo-treated groups 42,43 . Orlistat has been demonstrated to reduce the incidence of type 2 diabetes 41, 44 , improve diabetic control and insulin sensitivity 45-47 , and reduce cardiovascular risk factors 48, 49 . A...

Mechanisms of malnutrition

In Crohn's disease involving the small intestine, the diseased mucosa may cause maldigestion of nutrients. This can occur in proximal disease where mucosal disaccharidases are destroyed by mucosal injury, causing undigested carbohydrates to provide osmotic loads with resulting diarrhea and flatulence from metabolism by colonic flora. Additionally, ileal disease may result in decreased bile acid absorption, thus decreasing the bile acid pool. The result is that lipids are prevented from being solubilized, and thus they are poorly digested within the lumen of the proximal small bowel as a result of inadequate mixing of triglycerides with pancreatic enzymes. Furthermore, primary sclerosing cholangitis, a biliary complication of IBD, especially ulcerative colitis, may also result in decreased bile secretion and decreased digestion and absorption of fats and fat-soluble vitamins. Small intestinal bacterial

Bowel Retraining for Anal Incontinence

Pictures Procon For Bowel

Evacuation retraining can also be accomplished with the help of a continence plug. The anal plug (the Pro con device) (Figure 12-3.1) consists ofa catheter that is inserted in the rectum and held in place by a balloon. The catheter has a sensor that detects flatus and stool. The catheter is attached to a beeper that signals when the rectum is full, preventing seepage and allowing adequate time to reach a bathroom and evacuate.

Adverse Reactions Contraindications and Interactions

Black cohosh may cause nausea, vomiting, hypotension, and even miscarriage. It is absolutely contraindi-cated in pregnancy. Red clover contains coumarins and should therefore be avoided with anticoagulants. Diets high in red clover isoflavones have reduced livestock fertility and theoretically could do the same in humans. Flaxseed may cause nausea, diarrhea, and flatulence. Cyanogenic nitrates in flax (especially in immature seed pods) have produced toxic reactions.

Assessment of abdominal injury

There is a lower risk of serious visceral injury with stab wounds than with gunshot wounds. Less than 50 per cent require an emergency laparotomy. In the stable patient, local wound exploration can identify peritoneal perforation. If there is no violation of the peritoneum, discharge can be considered. Abdominal stab wounds with hypotension and abdominal distension, peritonitis, significant evisceration, or other clear signs of visceral injury require prompt laparotomy. Gunshot wounds are difficult to explore locally, and exploratory laparotomy is usually indicated.

Conservative management

Percutaneous peritoneal lavage (usually 1-2 l h of dialysis fluid or normal saline) should be considered early whenever dark brown ascitic fluid is obtained. Lavage is usually discontinued when the returned fluid is clear. Striking reduction of early cardiorespiratory dysfunction and early mortality has been demonstrated, in particular for alcohol-induced pancreatitis. These benefits are ascribed to the early removal of toxic substances before they reach the systemic circulation. Peritoneal lavage is also useful in patients with coexisting renal failure and or fluid overload. However, it seems unlikely that enough lavage fluid gains access to the lesser sac where the concentration of toxic agents must be greatest. This therapy neither slows progression of local tissue injury nor prevents the development of necrosis and infection, and thus fails to influence overall mortality. A dramatic reduction in the incidence and subsequent mortality from pancreatic infection has been obtained by...

Management of Complications

Aerophagia, or swallowing air, is frequently reported by patients, but rarely intolerable (122). Minor clinical signs are eructation, flatulence, and abdominal discomfort. Aerophagia is usually dependent on the level of inspiratory pressure and is more commonly seen when using volume and or mouthpiece ventilation and in the care of patients with neuromuscular disease. The incidence decreases if the peak inspiratory pressure is kept below 25 cm H2O pressure.


Increasing fiber foods in the diet may cause flatulence in some individuals because as complex carbohydrates are digested by bacteria in the intestine, methane gas is released. Eating smaller amounts frequently helps eliminate the problem so does selecting complex carbohydrates, through experimentation, that are better tolerated. Sometimes foods in combination cause gas while eating them singly does not. Ginger, garlic, peppermint, and fennel are carminatives or gas expellers and can be eaten or drunk as tea with or after meals. If a fiber supplement is necessary, a mucilaginous kind like psyllium husks or flaxseed is less irritating than crude bran fiber.


Acute gastric dilatation may complicate any condition associated with acute gastric retention. When it is severe, there is upper abdominal distension, hiccoughs are common, and patients who are not deeply sedated or unconscious usually have obvious discomfort and tachycardia. Copious vomiting or regurgitation is usually a late manifestation. The first indication of acute gastric dilatation in the critically ill is often a hugely distended stomach seen on a chest radiograph taken for other reasons.

C difficile

Pseudomembranous colitis presents with diarrhea and abdominal pain or discomfort. Tenesmus, anorexia, nausea, or vomiting may also occur. Flexible procto sigmoidoscopy may reveal the typical pseudomembranes, but their absence does not exclude the diagnosis up to a third of patients have pseudomembranes only in the ascending colon. Toxic megacolon is a rare complication of pseudomembranous colitis. Increasing abdominal pain, abdominal distension, fever, and tachycardia in a patient with recent diarrhea are clues to the diagnosis. Half the patients with toxic megacolon present with shock or hypotension and all are sufficiently ill to need admission to an ICU. Toxic megacolon has a high morbidity up to two-thirds of the patients may die despite active treatment.

Chronic infection

One study of US immigrants reported that the most common pathogens were Trichuris trichiura, Giardia lamblia, and Ascaris lumbricoides. Giardia lamblia was more prevalent in the younger than 5-year-old age group, and helminths were more prevalent in the 6- to 10-year-old age group. No helminths were found in immigrants who had been in the US for more than 3 years. Infection caused by intestinal parasites irritate the GI tract, cause pain, anorexia, flatulence, tenderness, and affect the host nutrition directly as a result of inflammatory and non-inflammatory diarrhea. Host response mechanisms include accelerated epithelial cell turnover 144 . Trace element deficiencies affect the host pathogen interaction. Examples include the exacerbating effect of selenium deficiency on Trypanosoma cruzi, which is responsible for Chagas disease 145 . Malnutrition can cause an imbalance in T cell subpopulations that may lead to a defective T cell maturation and a decreased specific anti-Ascaris IgE...


Protein is lost into the bowel lumen and the bowel wall, which becomes edematous. Distension of the small bowel stimulates secretion but does not enhance absorption. The contents of the large intestine may reflux through an incompetent ileocecal valve, resulting in bacterial overgrowth of the small bowel. The bowel contents produce gas which, if not discharged, leads to increased bowel dilatation. Flatus and defecation stop, leading to fecal impaction in the terminal colon and rectum.

Resistant Starch

The heat of cooking gelatinizes the starch granules, increasing their susceptibility to enzymic (a-amylase) breakdown. However, a proportion of the starch, called resistant starch (RS), is undigestible even after prolonged incubation with the enzyme. In cereals, RS represents 0.4 to 2 of the dry matter in potatoes, 1 to 3.5 and in legumes, 3.5 to 5.7 . RS has been categorized as the sum of the starch and degradation products not absorbed in the small intestine of a healthy person (3a). There are three main categories RS1, physically enclosed starch (partially milled grains and seeds) RS2, ungelatinized crystallite granules of the B-type x-ray pattern (as found in bananas and potatoes) and RS3, retrograded amylose (formed during the cooling of starch gelatinized by moist heating). Resistant starch escapes digestion in the small intestine, but it then enters the colon, where it can be fermented by the local resident bacteria (of which there are over 400...


Physical changes during pregnancy, including abdominal distension, fetal movement, bladder distention, urinary frequency, backache, and heartburn, all contribute to increased sleep fragmentation and decreased REM sleep. Weight gain may precipitate or worsen pre-existing sleep apnea. Conversely, increased minute ventilation, preference for the lateral sleep position, and decreased REM sleep time during pregnancy can decrease the risk for OSA (71).


In fructose malabsorption, ingestion of the ketohexose in quantity creates abdominal bloating, flatulence, and diarrhea. Persons with this condition appear to have a defect in fructose absorption. No assessments of intestinal GLUT 5 or its controlling gene have yet been made in any of these patients. If either glucose or galactose is ingested with fructose, fructose absorption is enhanced, and often no symptoms of malabsorption occur (50, 51).


Tenofovir is taken once daily and is generally well tolerated, perhaps because it produces less mitochondr-ial toxicity than the NRTIs. Nausea, vomiting, flatulence, and diarrhea occur in 10 or fewer patients. Resistance to tenofovir has been documented, and cross-resistance to NRTIs may occur.

Digestive aids

Promotes degradation of flatulence factors a-Galactosides are oligosaccharides present in plant matter, particularly in beans. They are not normally degraded in the human digestive tract due to the absence of an appropriate endogenous digestive enzyme (i.e. an a-galactosidase). However, upon their entry into the large intestine, these oligosaccharides are degraded by microbial al-6 galactosidases, thus stimulating microbial fermentation. The end-products of fermentation include volatile fatty acids, carbon dioxide, methane and hydrogen, which lead to flatulence. This can be avoided by minimizing dietary intake of food containing a-galactosides. Another approach entails the simultaneous ingestion of tablets containing a-galactosidase activity. If these 'flatulence factors' are degraded before or upon reaching the small intestine, then the monosaccharides released will be absorbed and, hence, will be subsequently unavailable to promote undesirable microbial fermentations in the large...

Nitrous oxide

Nitrous oxide should be avoided in patients with cerebral swelling as it increases cerebral blood flow and intracranial pressure. In patients with pre-existing pulmonary hypertension it increases pulmonary vascular resistance. As nitrous oxide is 34 times more soluble than nitrogen, it will diffuse into air-containing spaces more rapidly than nitrogen can diffuse out causing an increase in size of any air-containing space. Hence it should be administered with great care to patients with pneumothoraces, gut ileus because of abdominal distension, and atrial or ventricular septal defects because of the risk of air embolism from intravenous lines.


The liver not only processes nutrients but must detoxify all the harmful substances the villi were unable to prevent from being absorbed into the bloodstream. Other situations that can tax the liver considerably include overeating and eating foods that are refined. Refined foods are missing the nutrients they need to be properly metabolized. If the liver can no longer filter and cleanse the blood, or properly metabolize nutrients, or take care of its own health, it is because liver cells are damaged or begin to die. Liver damage is not easily detected by conventional testing and its condition may not be known until dysfunction becomes apparent through illness. Symptoms may range from headache, diarrhea, constipation, food sensitivities, flatulence, sleeplessness, and aching joints to cirrhosis and hepatitis.


Orlistat is a gastric and pancreatic lipase inhibitor which blocks the absorption of approximately one-third of the fat calories contained in a meal (37) which are lost in the stool. Diets containing 30 per cent or more calories from fat produce oily stools, flatulence with discharge, and faecal urgency. Orlistat has no discernible central nervous system effects.

Osmotics saline

Adequate renal function is needed to excrete excess magnesium. Patients who have renal insufficiency should avoid magnesium salts. Hypermagnesemia can result from continuous use of magnesium salts, causing symptoms such as drowsiness, weakness, paralysis, complete heart block, hypotension, flushing, and respiratory depression. Side effects from excess use are flatulence, diarrhea, abdominal cramps, nausea, and vomiting.

General principles

It is precisely because of this relatively delicate balance between the proper digestion of foods and the potential rotting of those foods that food combining principles are so important. If you research these concepts further on your own, you will also find that food combining has its very vocal critics. Of course, every health recommendation has its supporters and critics. I would advise you to let your body be your guide. If, after combining your foods properly, you find that your gas, belching, bloating, heartburn, and flatulence are lessening (or even disappearing ) for the first time in memory, I doubt that you'll be very interested in giving much consideration to what the critics of food combining have to say.

Frequent Symptoms

Presenting signs and symptoms of children with neu-roblastoma reflect both the location of the primary tumor and the extent of disease. The typical patient with neuroblastoma is an infant or a toddler. Pain from abdominal distension or metastases to bone is common (30 ). Many patients fail to gain weight or have weight loss (11 ). The characteristic bilater In contrast, infants with stage 4S commonly present with abdominal distension resulting from massive liver infiltration (Fig. 7.1 a) and subcutaneous nodules (most of them better palpable than visible, sometimes blueberry appearance Fig. 7.1b). The massive hepatomegaly can lead to respiratory distress, and kidney or bowel function can be impaired due to obstruction by the tumor. Their medical condition can rapidly deteriorate within hours or days (Berthold et al. 1990).


Frequent cause of chronic diarrhea in pediatric patients. Often associated with bloating and flatulence. Milk or milk products exacerbate the diarrhea. Congenital lactase deficiency is an autosomal-recessive disorder that presents in infancy and is extremely rare. Late-onset lactose intolerance is due to a progressive loss of enzyme activity in the brush border

Diloxanide Furoate

Diloxanide furoate (Furamide) is an amebicide that is effective against trophozoites in the intestinal tract. In mild or asymptomatic infections, cures of 83 to 95 have been achieved in patients with dysentery, cure rates may be less impressive. The drug is administered only orally and is rapidly absorbed from the gastrointestinal tract following hydrolysis of the ester group. It is remarkably free of side effects, but occasionally flatulence, abdominal distention, anorexia, nausea, vomiting, diarrhea, pruritus, and urticaria occur. Diloxanide is excreted in the urine, largely as the glucuronide. It is not available in the United States.

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