Spikes on the surface of virus particles function as adherence factors. Viruses, as a group, have evolved mechanisms to avoid the antiviral effects of interferon and to prevent apoptosis of the host cell. Damage is often due to the host response.
■ From a virus's perspective, why would it be beneficial to prevent apoptosis?
■ From a virus's perspective, why would it be beneficial to induce apoptosis?
■ Why would various unrelated viral infections often include a similar set of symptoms (fever, headache, fatigue, and runny nose)?
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