Aids

Slow developing disease. Transmissible agents "multiply" without resulting in disease symptoms until they cause a fatal infection.

Days

Time

Years

Neuron in root ganglion

Pons

Neuron in root ganglion

Pons

Vims moves up trigeminal nerve

Latency of herpesviruses

The initial Infection occurs in 3-5-year-old children and symptoms include cold sores or fever blisters. The virus then moves up the trigeminal nerve where it remains latent in the sensory ganglia of the nerve.

Vims moves up trigeminal nerve

Latency of herpesviruses

The initial Infection occurs in 3-5-year-old children and symptoms include cold sores or fever blisters. The virus then moves up the trigeminal nerve where it remains latent in the sensory ganglia of the nerve.

Activation of virus In neuron

The latent virus Is activated and cold sores can recur after the virus moves back down the nerve. The lesions are caused by one type of herpesvirus, herpes simplex type 1 (HSV-1).

Virions move down trigeminal nerve

The latent virus Is activated and cold sores can recur after the virus moves back down the nerve. The lesions are caused by one type of herpesvirus, herpes simplex type 1 (HSV-1).

Virions move down trigeminal nerve

Figure 14.13 Infection Cycle of Herpes Simplex Virus, HSV-1

14.3 Interactions of Animal Viruses with Their Hosts 355

and infectious virions are not produced. Some examples of viruses that cause chronic infections are summarized in table 14.8.

Replication of DNA is very unusual in the hepatitis B virus. Unlike all other double-stranded DNA viruses, which replicate their DNA using the viral DNA as a template, the hepatitis B virus synthesizes RNA from the DNA. The RNA is then used as a template for the synthesis of DNA, using a viral-encoded DNA polymerase. This enzyme acts as a reverse tran-scriptase, an unusual enzyme found only in the retroviruses, a group of viruses that will be discussed shortly. Replication can be simply diagrammed as shown here:

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