The reninangiotensinaldosterone system

Pressor response to angiotensin II and other vasoconstrictors is enhanced in pre-eclampsia. The renin-angiotensin-aldosterone system is activated in normal pregnancy but suppressed in pre-eclampsia. Low plasma renin activity may contribute to the reduced intravascular volume in pre-eclampsia. Reports of serum angiotensin II levels in pre-eclampsia compared with those in normal pregnancy are conflicting. Angiotensin receptors are upregulated in pre-eclampsia, and high circulating angiotensin II levels may mediate the marked increase in blood pressure in at least some women with this condition.

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