There is evidence that genetic factors, abnormal placentation, endothelial dysfunction, and neutrophil and platelet activation are involved in the pathophysiology of pre-eclampsia (Fig 1). Alterations in the renin-angiotensin-aldosterone system may also contribute.

Fig. 1 Proposed model of the pathophysiology of pre-eclampsia: STBM, syncytiotrophoblast basement membrane; CNS, central nervous system.

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