Edema after ischemic stroke

This description arises from vascular occlusion models in animals. The initial component after vascular occlusion is cytotoxic, owing to the energy depletion of membrane ion pumps which results in intracellular swelling, and is potentially reversible. After reperfusion vasogenic edema occurs due to biphasic opening of the blood-brain barrier. Autoregulation is lost during ischemia and reperfusion causes a short-lived breakdown of the blood-brain barrier sufficient to promote influx of vasoactive substances which subsequently lead to a permanent breakdown of the barrier ( BetzaQd.Crockard 1992).

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