- A - 2
- Abstract
- Acetate Deficiency Hypothesis Of Cd
- Acetate Deficiency In Cd Mice
- Acetyl Aspartate
- Acknowledgements
- Activitydependent Regulation Of Naag Expression
- Advances In Experimental Medicine And Biology
- Amyotrophic Lateral Sclerosis 275
- Aspa
- Aspa Gene Knockout In The Mouse
- Aspartate Transport In Glial Cells
- Aspartoacylase Assay
- Aspartoacylase Deficiency In Cd
- Aspartoacylase Gene
- Astroglial Cells
- B
- Cell Harvesting Extraction and Preparation of Samples
- Cellular Aspartoacylase Activity
- Cellular Localization Studies Of Aspa Vs
- Ci
- Colocalization
- Comparative NAA and NAAG Expression in Hippocampus
- Comparative NAA and NAAG Expression in Neocortex
- Comparative Phylogenetic Distribution
- Comparison of Tissue Aspartoacylase Activity and Developmental Evolution Pattern
- Concluding Remarks
- Conclusion - 2
- Conclusion And Future Directions
- Conclusions - 2 3 4 5 6
- Control Of Brain Volume During Hypoosmolality And Hyperosmolality
- D
- Native gel Sds Page
- Discussion - 2 3 4 5 6
- E
- External
- F
- Future Directions
- G
- G123e
- Gcp Ii Inhibition Is A Novel Mechanism For Reduction Of Excitotoxic Glutamate
- Gcp Ii Inhibitors Prevent Neurodegeneration
- Gcp Ii Inhibitors Provide Neuroprotection
- Gln
- H
- H244r
- Hyperosmolality
- Hyponatremia
- I
- 1
- Ii I I I I I I I I I I I I I I I I l 605040 PPm
- In Nonjewish Patients With Canavan Disease 165
- Info - 2 3 4 5 6 7 8 9 10 11 12 13 14 15 16 17 18 19 20 21
- Introduction - 2 3 4 5 6 7 8 9 10 11 12 13 14 15 16 17 18 19 20 21
- Sds Page
- L
- M
- Methods - 2
- Methods And Human Subjects 3 4 5
- Multiple Lines Of Evidence Indicating A Strong Relationship Between Naa And Myelin Lipid Synthesis
- N
- NAA and the Osmolyte Hypothesis
- NAA identification and quantification
- Naa Is A Surrogate Marker Of Neuronal Health
- Naa Naag and Aspartoacylase Expression in Corpus Callosum
- Naa Naag And Canavans Disease The First International Syposium On Aacetylaspartate
- Naa Naag and GAD67 Expression in Forebrain and Midbrain
- NAA Toxicity in Canavan Disease
- Naag
- NAAG and GAD67 Expression in Rhesus Monkey Motor Cortex
- NAAG and NAA in GABAergic Neuronal Groups
- NAAG Biosynthesis
- NAAG Expression in Rhesus Monkey Motor Cortex
- NAAG in Groups with Known Neurotransmitters
- Nerve Fiber
- O
- Other Regions
- Other Sensory Systems
- P - 2
- Pain Models
- Patients And Methods
- Phylogeny
- Pmpa And 2mppa Are Potent And Specific Gcp Ii Inhibitors
- Preclinical Efforts Toward Acetate Supplementation Therapy
- Preparation of Cortical Neurons
- Preparation of O2A Progenitor Cells
- Proton Magnetic Resonance Spectroscopy
- Purification Of Aspnat
- Q
- Question And Answer Session - 2 3 4 5 6 7 8 9 10 11 12 13 14
- Questions And Answer Session
- R
- Reduced Naa In
- References - 2 3 4 5 6 7 8 9 10 11 12 13 14 15 16 17 18
- Results - 2
- Results And Discussion 3 4 5 6 7
- S 3j6
- Springer
- Summary
- T
- Technical Aspects
- Therapeutic Approaches For Cd
- Tissue Harvest for Aspartoacylase Assays
- Traumatic Brain Injury
- W
- Wacetylaspartylglutamate In The Nervous System
- Wacetylaspartylglutamate Naag
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