Breast cancer affects around 1 in 12 women, and is the leading cause of death in females between the ages of 40 and 50 in the West world (1).
In the last decade a wealth of studies have indicated a link between the pathogenesis of breast cancer and the expression of cyclooxygenases, particularly cyclooxygenase-2 (COX-2).
Non-steroidal anti-inflammatory drugs (NSAIDs), well-tolerated, accessible, and inexpensive medications, suppress COX activity, leading to speculation of a role for NSAIDs in breast cancer treatment and prevention. Not surprisingly, the activity and implications of expression
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