Concluding Remarks

HGF and its partner c-Met play a definitive role in tumour-stromal interactions, leading to particularly invasive and metastatic cancers. The invasion and subsequent establishment of metastasis are devastating events for patients with cancer. Therapeutic strategies targeting the activation of HGF warrant investigation for their potential value in combating the spread of tumours. HAI-1 and HAI-2 are serine protease inhibitors that display unique therapeutic potential due to their ability inhibit HGFA and matriptase action, and thus prevent the generation of biologically active HGF. These inhibitors play important roles in controlling the aggressive nature and spread of cancer. Further progress will undoubtedly lead to the application of these advances in the generation of future therapies to prevent the spread of breast cancer.

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