Further reading

Side L, Taylor B, Cayouette M et al. (1997) Homozygous inactivation of the NF1 gene in bone marrow cells from children with neurofibromatosis type 1 and malignant myeloid disorders. New England Journal of Medicine, 336, 1713-1720.

Song WJ, Sullivan MG, Legare RD et al. (1999) Haploinsufficiency of CBFA2 causes familial thrombocytopenia with propensity to develop acute myelogenous leukaemia. Nature Genetics, 23, 166-175.

Sood R, Talwar-Trikha A, Chakrabarti SR et al. (1999) MDS1/EVI1 enhances TGF-beta1 signaling and strengthens its growth-inhibitory effect but the leukemia-associated fusion protein AML1/MDS1/EVI1, product of the t(3;21), abrogates growth-inhibition in response to TGF-beta1. Leukemia, 13, 348-357.

Tartaglia M, Niemeyer CM, Fragale A et al. (2003) Somatic mutations in PTPN11 in juvenile myelomonocytic leukemia, myelodysplastic syndromes and acute myeloid leukemia. Nature Genetics, 34, 148-150.

Uchida T, Kinoshita T, Nagai H et al. (1997) Hypermethylation of the p15INK4B gene in myelodysplastic syndromes. Blood, 90, 1403-1409.

Van den Berghe H, Cassiman JJ et al. (1974) Distinct haematological disorder with deletion of long arm of no. 5 chromosome. Nature, 251, 437-438.

Wattel E, Preudhomme C, Hecquet B et al. (1994) p53 mutations are associated with resistance to chemotherapy and short survival in hematologic malignancies. Blood, 84, 3148-3157.

Yoneda-Kato N, LookAT, Kirstein MN etal. (1996) The t(3;5)(q25.1;q34) of myelodysplastic syndrome and acute myeloid leukemia produces a novel fusion gene, NPM-MLF1. Oncogene, 12, 265-275.

Zhao N, Stoffel A, Wang PW et al. (1997) Molecular delineation of the smallest commonly deleted region of chromosome 5 in malignant myeloid diseases to 1-1.5 Mb and preparation of a PAC-based physical map. Proceedings of the National Academy of Sciences of the United States of America, 94, 6948-6953.

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