Neurochemical Models Of Depression

Brain neurochemistry was one of the first suspects in the search for the biological basis of depression. The original monoamine hypothesis of depression derived from the findings that monoamine depletion by the drug reserpine caused depression, while antidepressants inhibited monoamine reuptake. Subsequent reports that there were reduced monoamine breakdown products in the CSF led to the theory that there is a deficiency of noradrenaline, dopamine, 5-HT, or all three at monoaminergic synapses. From this original hypothesis, several proposed biological models of depression are worth discussing.

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